Cerebral Palsy: Causes and Treatments found to be Beneficial
Cerebral palsy (CP) is an umbrella term encompassing a group of non-progressive, non-contagious motor conditions that cause physical disability in human development, chiefly in the various areas of body movement.
Cerebral refers to the cerebrum, which is the affected area of the brain (although the disorder most likely involves connections between the cortex and other parts of the brain such as the cerebellum), and palsy refers to disorder of movement. Cerebral palsy is caused by damage to the motor control centers of the developing brain and can occur during pregnancy, during childbirth or after birth up to about age three.
Cerebral palsy describes a group of permanent disorders of the development of movement and posture, causing activity limitation, that are attributed to nonprogressive disturbances that occurred in the developing fetal or infant brain. The motor disorders of cerebral palsy are often accompanied by disturbances of sensation, perception, cognition, communication, and behaviour, by epilepsy, and by secondary musculoskeletal problems.
Spastic cerebral palsy is by far the most common type, occurring in 70% to 80% of all cases. Moreover, spastic CP accompanies one of the other types in 30% of all cases. People with this type are hypertonic and have a neuromuscular condition stemming from damage to the corticospinal tract or the motor cortex that affects the nervous system's ability to receive gamma amino butyric acid in the area(s) affected by the disability. Spastic CP is further classified by topography dependent on the region of the body affected;
Recent research has demonstrated that intrapartum asphyxia is not the most important cause, probably accounting for no more than 10 percent of all cases; rather, infections in the mother, even infections that are not easily detected, may triple the risk of the child developing the disorder, mainly as the result of the toxicity to the fetal brain of cytokines that are produced as part of the inflammatory response. Toxicity exposures (toxic metals or some antibiotics) to the mother can also be a factor. Low birthweight is a risk factor for CP—and premature infants usually have low birth weights, less than 2.0 kg, but full-term infants can also have low birth weights. Multiple-birth infants are also more likely than single-birth infants to be born early or with a low birth weight.
After birth, other causes include toxins, severe jaundice, mercury or lead poisoning, physical brain injury, shaken baby syndrome, incidents involving hypoxia to the brain (such as near drowning), and encephalitis or meningitis. The three most common causes of asphyxia in the young child are: choking on foreign objects such as toys and pieces of food, poisoning, and near drowning.
Adequate nutrition and growth play a protective role in the development of cerebral palsy. Nutritional counseling may help when dietary needs are not met because of problems with eating certain foods. Adequate mineral content has a profound effect on a child's IQ. Those given enriched formula had IQ readings 14 points higher than those on standard formula, and showed a lower incidence of cerebral palsy (BMJ 98;317:1981-1987). Pasamanick and Knobloch (1966) reviewed a large number of studies and concluded that toxemia and anoxia, cerebral palsy, epilepsy, mental retardation, behavioral disorganization, and reading disability are functionally related to dietary deficiency.
(Mannatech Ambrotose supplementation has been shown to have benefits)
Cases improved after Mannatech products : www.mannatech.com/
Cerebral Palsy Nancy Rossnets (Mannafest 05); Therapist (Mannafest 05); Karen Vogel (Portland 04);
mannatech community ID #673410 you need password to see the testimonials study: Arcadi V;. Case report: ataxic cerebral palsy multidisciplinary treatment effects including dietary supplementation. Proc Fisher Inst Med Res. 2002 Apr 1; 2: 7-9.
Both massage therapy and hatha yoga are designed to help relax tense muscles, strengthen muscles, and keep joints flexible. Hatha yoga breathing exercises are sometimes used to try to prevent lung infections.
"Everyone has always known that children with developmental problems - cerebral palsy, Down's Syndrome, and autism - have bowel problems." He believes that this is caused by 'funny nervous systems', possibly exacerbated by what he describes as 'abnormal diets': whether of their own choosing.
Cellulase deficiency is a malabsorption syndrome (impaired absorption of nutrients, vitamins, or minerals from the diet by the lining of the small intestine) with its many symptoms of lower abdominal gas, pain, bloating and problems associated with the jejunum and pancreas. Other conditions associated with cellulose deficiency include nervous system conditions such as Bell's Palsy, Tic and facial neuralgia, all of which respond remarkably to cellulase. Certain toxic conditions, such as chemicals, drugs and toxic metals, including silver amalgam fillings (mercury in the teeth) are greatly alleviated with cellulase. This also includes acute food allergies. People who have malabsorption syndrome and cellulose deficiency have a tendency toward sugar and/or gluten intolerance.
For a number of years the media has had us clambering for more fiber in our
diets. What we are not told is that the cellulase which breaks down the
fiber has been removed from those products. The soluble fiber forms a
gel-like matrix in the small and large intestine which delays the passage
of food through the colon, forming gas and other problems. This gel-like
substance creates malabsorption. L. Plantarum is an unusual lactobacillus
in that it liquefies gelatin.
Check out the enzyme page here.
Cerebral palsy shares some characteristics with autism, for which more information is available. Toxic metal toxicity and vaccination have been found to be major factors in most autism cases, and metals chelation has been documented by large numbers of clinical trials to be the most effective treatment of autism. www.flcv.com/kidshg.html
Medical mistakes are one cause of many cerebral palsy cases. Doctors and hospitals sometimes make mistakes during delivery. Birth mistakes usually occur right before, during, or after delivery. Indications that a medical mistake happened include:
the delivery was unduly delayed. the child was not breathing after birth.
the child had seizures after birth. the child was transferred to Intensive Care. the child had an MRI after birth.
A study of 4000 pregnant women carried out by the University of Leicester has found that children born to mothers given a combination of two antibiotics in an attempt to stop them from giving birth prematurely had an almost three fold higher risk of developing cerebral palsy,
The drugs each also increased the risk when given singly but to a lesser degree.
Children whose mothers had the antibiotics were also more likely
to have problems such as having to wear glasses, struggling to walk around the
block and difficulties with day to day problem solving at age seven. More... Sun,
Antibiotics used during pregnancy cause increased risk of cerebral palsy in newborns, The Lancet, Sept 2008.
According to a
study published in The Lancet, while the antibiotics erythromycin and
co-amoxiclav have an immediate effect on women in premature labor, they
increase the long-term risk of cerebral palsy in newborns.
Scientists believe that compared to the direct effects of antibiotics, the situation for which they have been prescribed are more likely to place newborns at a higher risk of developing cerebral palsy.
The administration of these antibiotics postpones labor and reduces the risk of infection and breathing problems in babies born to women whose waters have broken.
Cerebral palsy, a physical impairment affecting movement, is the result of development failure in part of the child's brain either before birth or in early childhood. Labor complications, prematurity along with prenatal and infantile infections and toxic exposures are the main causes of the condition.
Mercury Toxicity of Mother and/or Infant
exposure of humans to very high levels of methyl mercury results in
exposure to methylmercury is known to result in severe neurological effects on
the developing fetus and infant, including cerebral palsy, mental retardation,
and seizures. Males are more susceptible than females to neurological damage
from perinatal methylmercury exposures. Preliminary analyses of data and
statistics for the hospitalization rates of males for cerebral palsy in the 17
Canadian Areas of Concern in the
Ontario communities that have had large industrial releases of mercury have
hospitalization rates for cerebral palsy among boys and men that are up to five
times higher than the average in the rest of the province. Males in
was responsible for the first known epidemic of cerebral palsy from
a toxin, when it was dumped into
vinyl plastics factory (Textbook of Pediatrics 1996 WB Saunders). Minamata disease resulted from in-utero exposure to mercury-contaminated fish. Children with Minamata disease had symptoms indistinguishable from mental retardation or cerebral palsy (Kondo 2000). Individual susceptibility played an important role in both disorders. Although thousands of children were treated with mercury-containing teething powders, only one in 500 to one in 1,000 children who were exposed developed Acrodynia (Warkany 1966). The role of individual sensitivity made it extremely difficult to link mercury exposure with what was, at the time, a new and bizarre disease. Similarly, when children have been exposed to high levels of mercury in foods, only a small group develop severe mercury poisoning while thousands are apparently unharmed (Jalili 1961, Kondo 2000).
The motor deficits of cerebral palsy are usually unrecognizable before 4-6 months of age.
MERCURY: Influences on Body Chemistry http://www.drkaslow.com/html/mercury_s_influence.html
"Even when an injury occurs soon after a vaccination,
this may not immediately be noticeable. This applies generally to injuries of
the developing nervous system, regardless of the cause. Such neurological syndromes
as cerebral palsy and developmental language disorder may come to light months
or years after the brain damage was inflicted. The effects of severe injury may
take years to show up, for example as learning and attention problems."--Marcel
Kinsbourne, M.D. & says Tom Clarkson, a toxicologist at the
University of Rochester Medical Center.
It seems the days of Polio are still with us. Not in the form of
acute viral outbreaks of fever and paralysis, but in the unexplored statistics
on the long-term effects from the viral contaminated Polio vaccines given to
countless children and adults three decades ago. Some doctors are of the belief
that Polio has not been eradicated by vaccination, but cases now are under a
redefinition and new diagnostic names like Viral or Aseptic Meningitis. As the
recorded cases of "Polio" continued to decline, there was a
significant increase in "Cerebral palsy" or "Aseptic (Viral)
Meningitis" and "Guillain-Barré Syndrome.
For example, in
Mercury and other heavy metals (such as lead) can cause progressive myelin degeneration with the development of antibodies to myelin basic protein (MBA) and glial fibrillary acidic protein (GFAP). Recent discovery of herpes virus-6 in the damaged areas of the brains of 73% of Multiple Sclerosis sufferers is indeed disturbing. The nervous system, once the insulation is stripped, can be likened to your home with bare wires inside the walls—a dangerous situation. In the body, symptoms may be many and varied:
1) tremors, shaking, “palsy” due to malfunction of nerve transmissions
2) uncoordination in walking, writing, and other automatic physical movements
3) slurred speech
4) excessive salivation
5) deterioration of memory and thinking processes
6) blurred vision
7) difficulty urinating, incontinence
8) environmental sensitivity, allergic to smells, food, clothing, electrical equipment
9) breathing problems, short of breath
10) nervousness or nervous breakdown
11) numbness and tingling in extremities
12) heart problems/arrhythmias.
Some have found Sphingolin™ most helpful (Ecological Formulas 800-888-4585). Vitamin B12 is often lacking, and it is essential to sheath formation. Additionally, nervonic acid, EFAs, and very-long-chained-fatty alcohols have clinically been shown to yield positive outcomes. These benefit the myelin sheath, increasing perception and response. I think it better to address the need for HCl first. Low HCl production is associated with many problems.
Until the JAMA article, Dr. Reg McDaniel’s science team had been unable to explain how adults with Alzheimer’s, Parkinson’s, and Huntington’s, and children with cerebral palsy, leukodystrophy, Down’s syndrome, autism, and FAS experience restorations in brain function with the addition of glyconutrients and other micronutrients to their diet.
Prior to this knowledge in 2003 from JAMA, there was no reasonable, scientific explanation of how the many individuals with presumed permanent brain injury from strokes, trauma, or neurodegenerative disorders regain lost, central-nervous-system function that physicians and scientists had regarded as permanent and irreversible.
Our new understanding of glyconutrients stimulating the development of one’s bone-marrow stem cells, which have the capacity to develop into any cell the body needs, provides us with a scientific understanding of how such unparalleled restorations of brain function can be induced by dietary supplementation of glyconutrients, (especially when stimulated to activity by the growth factors of colostrum. Lithium orotate in a daily supplement of 30-68 mg is also known to effectively regenerate neurons. Taking a bit of fatty acid with the lithium will ensure against toxicity.)
Another case history involves Sara, a premature FAS child with heart defects and cerebral palsy at birth. Sara’s adoptive mother began giving her micronutrients daily by rubbing a cream with glyconutrients on her entire body in the premature nursery. When Sara could drink from a bottle, the adoptive mother gave her glyconutrients followed by all of the micronutrients in a bottle. When evaluated by pediatric specialists at 4 years old, she no longer had any evidence of FAS or cerebral palsy, and her heart developed normally and required no surgery.
Photographs of Sara show that at birth her face displayed the characteristics of a child with severe FAS. By age four her face had lost all FAS characteristics and appeared “normal” and her IQ changed from less than 80 to over 100. This is an unprecedented change for an FAS child. These individual case history reports do not provide scientific proof. However, they do give us hope and are leading to additional studies.